iga nephropathy will not be genetic it?

With iga nephropathy increasing year by year, many patients will consider iga nephropathy will not be genetic, so iga nephropathy in the end it will not be genetic?

Kidney disease is mainly due to the abnormal deposition of immunoglobulin iga mesangial area, which led to kidney disease. It is an immune system deficiency diseases, a lot of incentives, usually caused by recurrent inflammation, tonsillitis, colds and other causes.

Iga nephropathy only for himself kidney inherent cell damage, there will be no iga nephropathy genetic phenomenon.

The most common change iga nephropathy due to the different performance of different pathologies, optical microscopy confirmed that IgA nephropathy, mesangial area focal or diffuse expansion of extracellular matrix, and the expansion of mesangial cells and matrix by no means specific IgA nephropathy,

In many other kidney diseases also it can be observed that the above changes, these diseases include diabetic nephropathy, focal segmental glomerular mesangial sclerosis and many of the diseases associated with glomerular damage. Moreover, in patients with IgA nephropathy can be seen in a variety of the

Optical microscopy to determine the damage, including diffuse capillary endothelial proliferation, segmental sclerosis, segmental necrosis and cellular crescent formation.

Light microscopy lesions mainly involving the glomerular lesions varied types, including minor lesions, mesangial proliferative disease, focal segmental lesions, proliferative lesions within the capillaries, crescents and sclerosis lesions and other lesions, Most cases with diffuse mesangial

Proliferation typical changes, including mesangial cell proliferation and mesangial matrix increase. It can be further divided into mild, moderate and severe glomerular mesangial proliferative disease according to disease severity. By Masson staining often can be found in the mesangial area eosinophilic material deposition, often

Massive distribution in mesangial area. Slight lesions were only slight mesangial proliferation, also showed diffuse hyperplasia.


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