iga nephropathy inherited?

iga nephropathy hereditary it? iga nephropathy are many patients who watched a lot of people do not know whether genetic iga nephropathy, Can you understand it. "For this question, let us look at the experts describes it.

iga nephropathy inherited? Experts, iga nephropathy is not hereditary.

Kidney disease is mainly due to the abnormal deposition of immunoglobulin iga mesangial area, which led to kidney disease. It is an immune system deficiency diseases, a lot of incentives, usually caused by recurrent inflammation, tonsillitis, colds and other causes. Iga nephropathy only for himself kidney inherent cell damage, there will be no iga nephropathy genetic phenomenon.

Kidney pathology due to different conditions, and different manifestations:

The most common change in IgA nephropathy associated optical microscope confirmed that the mesangial area focal or diffuse expansion of extracellular matrix, the expansion of mesangial cells and matrix IgA nephropathy is by no means unique in many other kidney diseases also can be observed to the above changes, these diseases include diabetic nephropathy, focal segmental glomerular mesangial sclerosis and many of the diseases associated with glomerular damage. Moreover, in patients with IgA nephropathy can be seen in many of the optical microscope to determine the damage, including diffuse capillary endothelial proliferation, segmental sclerosis, segmental necrosis and cellular crescent formation.

Light microscopy lesions mainly involving the glomerular lesions varied types, including minor lesions, mesangial proliferative disease, focal segmental lesions, proliferative lesions within the capillaries, crescents and sclerosis lesions and other lesions, Most cases with diffuse mesangial proliferation typical changes, including mesangial cell proliferation and mesangial matrix increase. It can be further divided into mild, moderate and severe glomerular mesangial proliferative disease according to disease severity. By Masson staining often can be found addicted to red material deposited in the mesangial area in Iraq, often patchily distributed in mesangial area. Slight lesions were only slight mesangial proliferation, also showed diffuse hyperplasia.


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